IBF Instituto Biofisika
Centro (uo)
Publicaciones en las que colabora con Asier Etxaniz Iriondo (14)
2024
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The N-terminal region of the ATG8 autophagy protein LC3C is essential for its membrane fusion properties
International Journal of Biological Macromolecules, Vol. 262
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Vesicle-vesicle fusion promoted by the Atg8-family autophagy protein LC3C: relevance of the N-terminal region
Autophagy
2023
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Effect of ATG12–ATG5-ATG16L1 autophagy E3-like complex on the ability of LC3/GABARAP proteins to induce vesicle tethering and fusion
Cellular and Molecular Life Sciences, Vol. 80, Núm. 2
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Effects of a N-Maleimide-derivatized Phosphatidylethanolamine on the Architecture and Properties of Lipid Bilayers †
International Journal of Molecular Sciences, Vol. 24, Núm. 23
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Lipids in Mitochondrial Macroautophagy: Phase Behavior of Bilayers Containing Cardiolipin and Ceramide
International Journal of Molecular Sciences, Vol. 24, Núm. 6
2022
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Autophagy protein LC3C binding to phospholipid and interaction with lipid membranes
International Journal of Biological Macromolecules, Vol. 212, pp. 432-441
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Ceramide enhances binding of LC3/GABARAP autophagy proteins to cardiolipin-containing membranes
International Journal of Biological Macromolecules, Vol. 217, pp. 748-760
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LC3 subfamily in cardiolipin-mediated mitophagy: a comparison of the LC3A, LC3B and LC3C homologs
Autophagy, Vol. 18, Núm. 12, pp. 2985-3003
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Phase behaviour of C18-N-acyl sphingolipids, the prevalent species in human brain
Colloids and Surfaces B: Biointerfaces, Vol. 219
2021
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Molecular and mesoscopic geometries in autophagosome generation. A review
Biochimica et Biophysica Acta - Biomembranes, Vol. 1863, Núm. 12
2020
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Irreversible versus repairable membrane poration: differences in permeabilization elicited by Bordetella Adenylate Cyclase Toxin and its hemolysin domain in macrophages
FEBS Journal, Vol. 287, Núm. 9, pp. 1798-1815
2018
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Membrane repair mechanisms against permeabilization by pore-forming toxins
Toxins, Vol. 10, Núm. 6
2017
2015
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Adenylate Cyclase Toxin promotes bacterial internalisation into non phagocytic cells
Scientific Reports, Vol. 5