Publicaciones en colaboración con investigadores/as de Ludwig Maximilian University of Munich (13)

2022

  1. Microglia states and nomenclature: A field at its crossroads

    Neuron, Vol. 110, Núm. 21, pp. 3458-3483

2021

  1. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

  2. Reactive astrocyte nomenclature, definitions, and future directions

    Nature Neuroscience, Vol. 24, Núm. 3, pp. 312-325

2020

  1. P2X7 Receptor-Dependent Layer-Specific Changes in Neuron-Microglia Reactivity in the Prefrontal Cortex of a Phencyclidine Induced Mouse Model of Schizophrenia

    Frontiers in Molecular Neuroscience, Vol. 13

2019

  1. Perspective: Of mice and Men – How widespread is adult neurogenesis?

    Frontiers in Neuroscience, Vol. 13, Núm. AUG

2018

  1. Crosslink between calcium and sodium signalling

    Experimental Physiology, Vol. 103, Núm. 2, pp. 157-169

2016

  1. Astrocytic Insulin Signaling Couples Brain Glucose Uptake with Nutrient Availability

    Cell, Vol. 166, Núm. 4, pp. 867-880

  2. Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

    Autophagy

2010

  1. Ca2+ imaging of intracellular organelles: Endoplasmic reticulum

    Neuromethods, Vol. 43, pp. 147-167

2008

  1. Inter-laboratory comparison of neuropathological assessments of β-amyloid protein: A study of the BrainNet Europe consortium

    Acta Neuropathologica, Vol. 115, Núm. 5, pp. 533-546

2007

  1. Occurrence and co-localization of amyloid β-protein and apolipoprotein E in perivascular drainage channels of wild-type and APP-transgenic mice

    Neurobiology of Aging, Vol. 28, Núm. 8, pp. 1221-1230

2006

  1. Selective vulnerability of different types of commissural neurons for amyloid β-protein-induced neurodegeneration in APP23 mice correlates with dendritic tree morphology

    Brain, Vol. 129, Núm. 11, pp. 2992-3005

2005

  1. Apolipoprotein E co-localizes with newly formed amyloid β-protein (Aβ) deposits lacking immunoreactivity against N-terminal epitopes of Aβ in a genotype-dependent manner

    Acta Neuropathologica, Vol. 110, Núm. 5, pp. 459-471