Publicaciones en colaboración con investigadores/as de Instituto de Salud Carlos III (36)

2024

  1. BirthSeq, a new method to isolate and analyze dated cells in different vertebrates

    Development (Cambridge), Vol. 151, Núm. 13

2022

  1. Genetically modified macrophages accelerate myelin repair

    EMBO Molecular Medicine, Vol. 14, Núm. 8

2021

  1. A proteomic approach for systematic mapping of substrates of human deubiquitinating enzymes

    International Journal of Molecular Sciences, Vol. 22, Núm. 9

  2. Future avenues for Alzheimer's disease detection and therapy: liquid biopsy, intracellular signaling modulation, systems pharmacology drug discovery

    Neuropharmacology, Vol. 185

  3. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

  4. Small GTPases of the Ras superfamily and glycogen phosphorylase regulation in T cells

    Small GTPases, Vol. 12, Núm. 2, pp. 106-113

  5. Using time causal quantifiers to characterize sleep stages

    Chaos, Solitons and Fractals, Vol. 146

2020

  1. AMPK-Dependent Mechanisms but Not Hypothalamic Lipid Signaling Mediates GH-Secretory Responses to GHRH and Ghrelin

    Cells, Vol. 9, Núm. 9

  2. Dendritic Cells and Microglia Have Non-redundant Functions in the Inflamed Brain with Protective Effects of Type 1 cDCs

    Cell Reports, Vol. 33, Núm. 3

  3. Glucose metabolism links astroglial mitochondria to cannabinoid effects

    Nature, Vol. 583, Núm. 7817, pp. 603-608

  4. LUZP1, a novel regulator of primary cilia and the actin cytoskeleton, is a contributing factor in townes-brocks syndrome

    eLife, Vol. 9, pp. 1-84

  5. Sex differences and gonadal hormone regulation of brain cardiolipin, a key mitochondrial phospholipid

    Journal of Neuroendocrinology, Vol. 32, Núm. 1

2019

  1. Elevated levels of Secreted-Frizzled-Related-Protein 1 contribute to Alzheimer’s disease pathogenesis

    Nature Neuroscience, Vol. 22, Núm. 8, pp. 1258-1268

  2. PARL deficiency in mouse causes Complex III defects, coenzyme Q depletion, and Leigh-like syndrome

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 116, Núm. 1, pp. 277-286

2018

  1. Differential effects of FXR or TGR5 activation in cholangiocarcinoma progression

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 4, pp. 1335-1344

  2. Erratum: Author Correction: Excitotoxic inactivation of constitutive oxidative stress detoxification pathway in neurons can be rescued by PKD1 (Nature communications (2017) 8 1 (2275))

    Nature communications

  3. In vivo imaging of Α7 nicotinic receptors as a novel method to monitor neuroinflammation after cerebral ischemia

    GLIA, Vol. 66, Núm. 8, pp. 1611-1624

  4. Role of the endocannabinoid system in drug addiction

    Biochemical Pharmacology, Vol. 157, pp. 108-121

  5. Singular Location and Signaling Profile of Adenosine A2A-Cannabinoid CB1 Receptor Heteromers in the Dorsal Striatum

    Neuropsychopharmacology, Vol. 43, Núm. 5, pp. 964-977

  6. Synaptic regulator α-synuclein in dopaminergic fibers is essentially required for the maintenance of subependymal neural stem cells

    Journal of Neuroscience, Vol. 38, Núm. 4, pp. 814-825