Asier
Larrea Sebal
Instituto Biofisika
Lejona, EspañaPublications in collaboration with researchers from Instituto Biofisika (14)
2024
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Evaluation of Various Types of Alginate Inks for Light-Mediated Extrusion 3D Printing
Polymers, Vol. 16, Núm. 7
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Extrusion-Based 3D Printing of Photocrosslinkable Chitosan Inks
Gels, Vol. 10, Núm. 2
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Multifunctional curcumin-based polymer coating: A promising platform against bacteria, inflammation and coagulation
Colloids and Surfaces B: Biointerfaces, Vol. 241
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OptiMo-LDLr: An Integrated In Silico Model with Enhanced Predictive Power for LDL Receptor Variants, Unraveling Hot Spot Pathogenic Residues
Advanced Science, Vol. 11, Núm. 13
2023
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Contribution of APOE Genetic Variants to Dyslipidemia
Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 43, Núm. 6, pp. 1066-1077
2022
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Boosting Cholesterol Efflux from Foam Cells by Sequential Administration of rHDL to Deliver MicroRNA and to Remove Cholesterol in a Triple-Cell 2D Atherosclerosis Model
Small, Vol. 18, Núm. 13
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Leu22_Leu23 Duplication at the Signal Peptide of PCSK9 Promotes Intracellular Degradation of LDLr and Autosomal Dominant Hypercholesterolemia
Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 42, Núm. 7, pp. E203-E216
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Lipoprotein(a) in hereditary hypercholesterolemia: Influence of the genetic cause, defective gene and type of mutation
Atherosclerosis, Vol. 349, pp. 211-218
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Pathophysiology of Atherosclerosis
International Journal of Molecular Sciences, Vol. 23, Núm. 6
2021
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MLb-LDLr: A Machine Learning Model for Predicting the Pathogenicity of LDLr Missense Variants
JACC: Basic to Translational Science, Vol. 6, Núm. 11, pp. 815-827
2020
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Cholesterol efflux efficiency of reconstituted HDL is affected by nanoparticle lipid composition
Biomedicines, Vol. 8, Núm. 10
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Pathophysiology of type 2 diabetes mellitus
International Journal of Molecular Sciences, Vol. 21, Núm. 17, pp. 1-34
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Statin treatment-induced development of type 2 diabetes: From clinical evidence to mechanistic insights
International Journal of Molecular Sciences, Vol. 21, Núm. 13, pp. 1-25
2019
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The Arg499His gain-of-function mutation in the C-terminal domain of PCSK9
Atherosclerosis, Vol. 289, pp. 162-172