Instituto Cajal -ko ikertzaileekin lankidetzan egindako argitalpenak (16)

2012

  1. Surface Expression and Subunit Specific Control of Steady Protein Levels by the Kv7.2 Helix A-B Linker

    PLoS ONE, Vol. 7, Núm. 10

2008

  1. A novel KCNQ4 pore-region mutation (p.G296S) causes deafness by impairing cell-surface channel expression

    Human Genetics, Vol. 123, Núm. 1, pp. 41-53

2004

  1. New roles of myosin II during vesicle transport and fusion in chromaffin cells

    Journal of Biological Chemistry, Vol. 279, Núm. 26, pp. 27450-27457

  2. Three mechanisms underlie KCNQ2/3 heteromeric potassium M-channel potentiation

    Journal of Neuroscience, Vol. 24, Núm. 41, pp. 9146-9152

2003

  1. Localization of KCNQ5 in the normal and epileptic human temporal neocortex and hippocampal formation

    Neuroscience, Vol. 120, Núm. 2, pp. 353-364

2002

  1. The identification and characterization of a noncontinuous calmodulin-binding site in noninactivating voltage-dependent KCNQ potassium channels

    Journal of Biological Chemistry, Vol. 277, Núm. 32, pp. 28545-28553

2001

  1. Intersubunit cooperativity in the NMDA receptor

    Neuron, Vol. 32, Núm. 6, pp. 1085-1096

2000

  1. Four residues of the extracellular N-terminal domain of the NR2A subunit control high-affinity Zn2+ binding to NMDA receptors

    Neuron, Vol. 25, Núm. 3, pp. 683-694

1999

  1. Protection by-imidazol(ine) drugs and agmatine of glutamate-induced neurotoxicity in cultured cerebellar granule cells through blockade of NMDA receptor

    British Journal of Pharmacology, Vol. 127, Núm. 6, pp. 1317-1326

1998

  1. Glycine-independent NMDA receptor desensitization: Localization of structural determinants

    Neuron, Vol. 20, Núm. 2, pp. 329-339

1997

  1. Desensitization of NMDA receptors as a mechanism of neuroprotection

    Methods and Findings in Experimental and Clinical Pharmacology

  2. Nonstationary noise analysis of M currents simulated and recorded in PC12 cells

    Journal of Neurophysiology, Vol. 77, Núm. 4, pp. 2131-2138

1996

  1. Inhibition of the Kv4 (Shal) family of transient K+ currents by arachidonic acid

    Journal of Neuroscience, Vol. 16, Núm. 8, pp. 2522-2531

  2. Inhibition of the Kv4 (Shal) family of transient K+ currents by arachidonic acid

    Journal of Neuroscience, Vol. 16, Núm. 3, pp. 1016-1025

  3. M-Current suppression in PC12 cells by bradykinin is mediated by a pertussis toxin-insensitive G-protein and modulated by intracellular calcium

    Brain Research, Vol. 740, Núm. 1-2, pp. 227-233

  4. Progression of M-current inhibition after agonist removal

    Journal of Neurophysiology, Vol. 75, Núm. 1, pp. 518-521