Publicaciones (19) Publicaciones de Asier Ruiz Nuñez

2023

  1. Hepatic levels of S-adenosylmethionine regulate the adaptive response to fasting

    Cell Metabolism, Vol. 35, Núm. 8, pp. 1373-1389.e8

2022

  1. Amyloid β / PKC-dependent alterations in NMDA receptor composition are detected in early stages of Alzheimer´s disease

    Cell Death and Disease, Vol. 13, Núm. 3

  2. GABA Receptor Agonists Protect From Excitotoxic Damage Induced by AMPA in Oligodendrocytes

    Frontiers in Pharmacology, Vol. 13

  3. Quercetin alleviates styrene oxide-induced cytotoxicity in cortical neurons in vitro via modulation of oxidative stress and apoptosis

    Drug and Chemical Toxicology, Vol. 45, Núm. 4, pp. 1634-1643

2021

  1. Polyphenols attenuate mitochondrial dysfunction induced by amyloid peptides

    Mitochondrial Physiology and Vegetal Molecules: Therapeutic Potential of Natural Compounds on Mitochondrial Health (Elsevier), pp. 317-337

  2. Role of Mitochondrial Dynamics in Microglial Activation and Metabolic Switch

    ImmunoHorizons, Vol. 5, Núm. 8, pp. 615-626

2020

  1. Mitochondrial division inhibitor 1 disrupts oligodendrocyte Ca2+ homeostasis and mitochondrial function

    GLIA, Vol. 68, Núm. 9, pp. 1743-1756

  2. Sephin1 protects neurons against excitotoxicity independently of the integrated stress response

    International Journal of Molecular Sciences, Vol. 21, Núm. 17, pp. 1-11

2018

  1. Blockade and knock-out of CALHM1 channels attenuate ischemic brain damage

    Journal of Cerebral Blood Flow and Metabolism, Vol. 38, Núm. 6, pp. 1060-1069

  2. Inhibition of Casein Kinase 2 Protects Oligodendrocytes From Excitotoxicity by Attenuating JNK/p53 Signaling Cascade

    Frontiers in Molecular Neuroscience, Vol. 11

  3. Mangiferin and morin attenuate oxidative stress, mitochondrial dysfunction, and neurocytotoxicity, induced by Amyloid Beta Oligomers

    Oxidative Medicine and Cellular Longevity, Vol. 2018

  4. Mitochondrial division inhibitor 1 (Mdivi-1) protects neurons against excitotoxicity through the modulation of mitochondrial function and intracellular Ca2+ signaling

    Frontiers in Molecular Neuroscience, Vol. 11

  5. Re-examining the potential of targeting ABHD6 in multiple sclerosis: Efficacy of systemic and peripherally restricted inhibitors in experimental autoimmune encephalomyelitis

    Neuropharmacology, Vol. 141, pp. 181-191

2014

  1. CGP37157, an inhibitor of the mitochondrial Na+/Ca2+ exchanger, protects neurons from excitotoxicity by blocking voltage-gated Ca2+ channels

    Cell Death and Disease, Vol. 5, Núm. 4

  2. Calcium dyshomeostasis in white matter injury

    White Matter Injury in Stroke and CNS Disease (Springer New York), pp. 433-460

  3. Measuring baseline Ca2+ levels in subcellular compartments using genetically engineered fluorescent indicators

    Methods in Enzymology (Academic Press Inc.), pp. 47-72

2013

  1. 1–42 β-amyloid peptide requires PDK1/nPKC/Rac 1 pathway to induce neuronal death

    Translational Psychiatry, Vol. 3, Núm. 1

2010

  1. Intracellular Ca2+ release through ryanodine receptors contributes to AMPA receptor-mediated mitochondrial dysfunction and ER stress in oligodendrocytes

    Cell Death and Disease, Vol. 1, Núm. 7

2009

  1. Endoplasmic reticulum Ca2+ release through ryanodine and IP3 receptors contributes to neuronal excitotoxicity

    Cell Calcium, Vol. 46, Núm. 4, pp. 273-281