Ion Signaling and Disease in Excitable Membranes Laboratory
Instituto Cajal
Madrid, EspañaPublicaciones en colaboración con investigadores/as de Instituto Cajal (16)
2012
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Surface Expression and Subunit Specific Control of Steady Protein Levels by the Kv7.2 Helix A-B Linker
PLoS ONE, Vol. 7, Núm. 10
2008
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A novel KCNQ4 pore-region mutation (p.G296S) causes deafness by impairing cell-surface channel expression
Human Genetics, Vol. 123, Núm. 1, pp. 41-53
2004
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New roles of myosin II during vesicle transport and fusion in chromaffin cells
Journal of Biological Chemistry, Vol. 279, Núm. 26, pp. 27450-27457
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Three mechanisms underlie KCNQ2/3 heteromeric potassium M-channel potentiation
Journal of Neuroscience, Vol. 24, Núm. 41, pp. 9146-9152
2003
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Localization of KCNQ5 in the normal and epileptic human temporal neocortex and hippocampal formation
Neuroscience, Vol. 120, Núm. 2, pp. 353-364
2002
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The identification and characterization of a noncontinuous calmodulin-binding site in noninactivating voltage-dependent KCNQ potassium channels
Journal of Biological Chemistry, Vol. 277, Núm. 32, pp. 28545-28553
2001
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Intersubunit cooperativity in the NMDA receptor
Neuron, Vol. 32, Núm. 6, pp. 1085-1096
2000
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Four residues of the extracellular N-terminal domain of the NR2A subunit control high-affinity Zn2+ binding to NMDA receptors
Neuron, Vol. 25, Núm. 3, pp. 683-694
1999
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Protection by-imidazol(ine) drugs and agmatine of glutamate-induced neurotoxicity in cultured cerebellar granule cells through blockade of NMDA receptor
British Journal of Pharmacology, Vol. 127, Núm. 6, pp. 1317-1326
1998
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Glycine-independent NMDA receptor desensitization: Localization of structural determinants
Neuron, Vol. 20, Núm. 2, pp. 329-339
1997
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Desensitization of NMDA receptors as a mechanism of neuroprotection
Methods and Findings in Experimental and Clinical Pharmacology
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Nonstationary noise analysis of M currents simulated and recorded in PC12 cells
Journal of Neurophysiology, Vol. 77, Núm. 4, pp. 2131-2138
1996
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Inhibition of the Kv4 (Shal) family of transient K+ currents by arachidonic acid
Journal of Neuroscience, Vol. 16, Núm. 3, pp. 1016-1025
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Inhibition of the Kv4 (Shal) family of transient K+ currents by arachidonic acid
Journal of Neuroscience, Vol. 16, Núm. 8, pp. 2522-2531
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M-Current suppression in PC12 cells by bradykinin is mediated by a pertussis toxin-insensitive G-protein and modulated by intracellular calcium
Brain Research, Vol. 740, Núm. 1-2, pp. 227-233
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Progression of M-current inhibition after agonist removal
Journal of Neurophysiology, Vol. 75, Núm. 1, pp. 518-521