Datos de investigación
Grupo: Endocrinología, Diabetes, Nutrición, Alteraciones Renales y Salud Infantil
18 datos de investigación referenciados
2024
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Additional file 10 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 10 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 4 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 4 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 5 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 5 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 6 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 6 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 7 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 7 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 8 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Additional file 8 of Sex bias in celiac disease: XWAS and monocyte eQTLs in women identify TMEM187 as a functional candidate gene
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Source Data Files from publication: A Protein Misfolding Shaking Amplification-based method for the spontaneous generation of hundreds of infectious prions, Nature Communications 2024
Zenodo
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Source Data Files from publication: A Protein Misfolding Shaking Amplification-based method for the spontaneous generation of hundreds of infectious prions, Nature Communications 2024
Zenodo