Publikationen in Zusammenarbeit mit Forschern von University of Bonn (22)

2021

  1. Future avenues for Alzheimer's disease detection and therapy: liquid biopsy, intracellular signaling modulation, systems pharmacology drug discovery

    Neuropharmacology, Vol. 185

  2. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

2019

  1. Elevated levels of Secreted-Frizzled-Related-Protein 1 contribute to Alzheimer’s disease pathogenesis

    Nature Neuroscience, Vol. 22, Núm. 8, pp. 1258-1268

2018

  1. Loss of Nucleobindin-2 Causes Insulin Resistance in Obesity without Impacting Satiety or Adiposity

    Cell Reports, Vol. 24, Núm. 5, pp. 1085-1092.e6

2016

  1. Astrocytes: a central element in neurological diseases

    Acta Neuropathologica, Vol. 131, Núm. 3, pp. 323-345

  2. Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

    Autophagy

2015

  1. Neuroinflammation in Alzheimer's disease

    The Lancet Neurology, Vol. 14, Núm. 4, pp. 388-405

2013

  1. Janus-faced microglia: Beneficial and detrimental consequences of microglial phagocytosis

    Frontiers in Cellular Neuroscience

2012

  1. Dispersible amyloid β-protein oligomers, protofibrils, and fibrils represent diffusible but not soluble aggregates: Their role in neurodegeneration in amyloid precursor protein (APP) transgenic mice

    Neurobiology of Aging, Vol. 33, Núm. 11, pp. 2641-2660

  2. Glial cells in (patho)physiology

    Journal of Neurochemistry, Vol. 121, Núm. 1, pp. 4-27

2009

  1. Capillary cerebral amyloid angiopathy is associated with vessel occlusion and cerebral blood flow disturbances

    Neurobiology of Aging, Vol. 30, Núm. 12, pp. 1936-1948

2008

  1. Glial cells are born with synapses

    FASEB Journal, Vol. 22, Núm. 8, pp. 2957-2969

  2. Inter-laboratory comparison of neuropathological assessments of β-amyloid protein: A study of the BrainNet Europe consortium

    Acta Neuropathologica, Vol. 115, Núm. 5, pp. 533-546

  3. Tracing of temporo-entorhinal connections in the human brain: Cognitively impaired argyrophilic grain disease cases show dendritic alterations but no axonal disconnection of temporo-entorhinal association neurons

    Acta Neuropathologica, Vol. 115, Núm. 2, pp. 175-183

2006

  1. Selective vulnerability of different types of commissural neurons for amyloid β-protein-induced neurodegeneration in APP23 mice correlates with dendritic tree morphology

    Brain, Vol. 129, Núm. 11, pp. 2992-3005

  2. The development of amyloid beta protein deposits in the aged brain.

    Science of aging knowledge environment : SAGE KE, Vol. 2006, Núm. 6

2005

  1. Apolipoprotein E co-localizes with newly formed amyloid β-protein (Aβ) deposits lacking immunoreactivity against N-terminal epitopes of Aβ in a genotype-dependent manner

    Acta Neuropathologica, Vol. 110, Núm. 5, pp. 459-471

2003

  1. Functional specialization of presynaptic Cav2.3 Ca2+ channels

    Neuron, Vol. 39, Núm. 3, pp. 483-496

2001

  1. Effect of 5-hydroxytryptamine 1A receptor agonist BAY X 3702 on BCL-2 and BAX proteins level in the ipsilateral cerebral cortex of rats after transient focal ischaemia

    Neuroscience, Vol. 107, Núm. 3, pp. 405-413

  2. Inhibition of store-operated Ca2+ influx by acidic extracellular pH in cultured human microglia

    GLIA, Vol. 36, Núm. 1, pp. 22-30