José Antonio
Rodríguez Pérez
![Foto de José Antonio](/img/nophoto.png)
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VU University Medical Center
Ámsterdam, HolandaPublicacións en colaboración con investigadores/as de VU University Medical Center (43)
2016
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A cellular reporter to evaluate CRM1 nuclear export activity: functional analysis of the cancer-related mutant E571K
Cellular and Molecular Life Sciences, Vol. 73, Núm. 24, pp. 4685-4699
2013
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USP1 deubiquitinase: Cellular functions, regulatory mechanisms and emerging potential as target in cancer therapy
Molecular Cancer, Vol. 12, Núm. 1
2009
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Combined assessment of EGFR pathway-related molecular markers and prognosis of NSCLC patients
British Journal of Cancer, Vol. 100, Núm. 1, pp. 145-152
2008
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Apoptosis pathways and new anticancer agents
Principles of Molecular Oncology: Third Edition (Humana Press), pp. 257-268
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Genotype analysis of the VNTR polymorphism in the SMYD3 histone methyltransferase gene: Lack of correlation with the level of histone H3 methylation in NSCLC tissues or with the risk of NSCLC
International Journal of Cancer
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Integration of gene dosage and gene expression in non-small cell lung cancer, identification of HSP90 as potential target
PLoS ONE, Vol. 3, Núm. 3
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Molecular mechanisms underlying the synergistic interaction of erlotinib, an epidermal growth factor receptor tyrosine kinase inhibitor, with the multitargeted antifolate pemetrexed in non-small-cell lung cancer cells
Molecular Pharmacology, Vol. 73, Núm. 4, pp. 1290-1300
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Role of cMET expression in non-small-cell lung cancer patients treated with EGFR tyrosine kinase inhibitors
Annals of Oncology, Vol. 19, Núm. 9, pp. 1605-1612
2007
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Bortezomib, but not cisplatin, induces mitochondria-dependent apoptosis accompanied by up-regulation of noxa in the non-small cell lung cancer cell line NCl-H460
Molecular Cancer Therapeutics, Vol. 6, Núm. 3, pp. 1046-1053
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EGFR and K-ras mutation analysis in non-small cell lung cancer: Comparison of paraffin embedded versus frozen specimens
Cellular Oncology, Vol. 29, Núm. 3, pp. 257-264
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Epidermal growth factor receptor (EGFR) gene copy number detection in non-small-cell lung cancer; a comparison of fluorescence in situ hybridization and chromogenic in situ hybridization
Histopathology, Vol. 51, Núm. 5, pp. 631-637
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Functional analysis of cancer-associated EGFR mutants using a cellular assay with YFP-tagged EGFR intracellular domain
Molecular Cancer, Vol. 6
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Genetic heterogeneity in patients with multiple neoplastic lung lesions: A report of three cases
Journal of Thoracic Oncology, Vol. 2, Núm. 1, pp. 12-21
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Homodimerization antagonizes nuclear export of Survivin
Traffic, Vol. 8, Núm. 11, pp. 1495-1502
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Immunohistochemical detection of nuclear survivin in NSCLC: A comparison of commercial antibodies [6]
Histopathology
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Nuclear localization of Survivin renders HeLa tumor cells more sensitive to apoptosis by induction of p53 and Bax
Cancer Letters, Vol. 250, Núm. 2, pp. 177-193
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Role of XIAP in inhibiting cisplatin-induced caspase activation in non-small cell lung cancer cells: A small molecule Smac mimic sensitizes for chemotherapy-induced apoptosis by enhancing caspase-3 activation
Experimental Cell Research, Vol. 313, Núm. 6, pp. 1215-1224
2006
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Enhanced cytotoxicity induced by gefitinib and specific inhibitors of the Ras or phosphatidyl inositol-3 kinase pathways in non-small cell lung cancer cells
International Journal of Cancer, Vol. 118, Núm. 1, pp. 209-214
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Erlotinib for frontline treatment of advanced non-small cell lung cancer: A phase II study
Clinical Cancer Research, Vol. 12, Núm. 20 PART 1, pp. 6049-6055
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Expression and localization of inhibitor of apoptosis proteins in normal human tissues
Human Pathology, Vol. 37, Núm. 1, pp. 78-86