Publicacions en què col·labora amb Asier Ruiz Nuñez (11)

2022

  1. Amyloid β / PKC-dependent alterations in NMDA receptor composition are detected in early stages of Alzheimer´s disease

    Cell Death and Disease, Vol. 13, Núm. 3

2021

  1. Polyphenols attenuate mitochondrial dysfunction induced by amyloid peptides

    Mitochondrial Physiology and Vegetal Molecules: Therapeutic Potential of Natural Compounds on Mitochondrial Health (Elsevier), pp. 317-337

2020

  1. Mitochondrial division inhibitor 1 disrupts oligodendrocyte Ca2+ homeostasis and mitochondrial function

    GLIA, Vol. 68, Núm. 9, pp. 1743-1756

  2. Sephin1 protects neurons against excitotoxicity independently of the integrated stress response

    International Journal of Molecular Sciences, Vol. 21, Núm. 17, pp. 1-11

2018

  1. Mangiferin and morin attenuate oxidative stress, mitochondrial dysfunction, and neurocytotoxicity, induced by Amyloid Beta Oligomers

    Oxidative Medicine and Cellular Longevity, Vol. 2018

  2. Mitochondrial division inhibitor 1 (Mdivi-1) protects neurons against excitotoxicity through the modulation of mitochondrial function and intracellular Ca2+ signaling

    Frontiers in Molecular Neuroscience, Vol. 11

2014

  1. CGP37157, an inhibitor of the mitochondrial Na+/Ca2+ exchanger, protects neurons from excitotoxicity by blocking voltage-gated Ca2+ channels

    Cell Death and Disease, Vol. 5, Núm. 4

  2. Calcium dyshomeostasis in white matter injury

    White Matter Injury in Stroke and CNS Disease (Springer New York), pp. 433-460

2013

  1. 1–42 β-amyloid peptide requires PDK1/nPKC/Rac 1 pathway to induce neuronal death

    Translational Psychiatry, Vol. 3, Núm. 1

2010

  1. Intracellular Ca2+ release through ryanodine receptors contributes to AMPA receptor-mediated mitochondrial dysfunction and ER stress in oligodendrocytes

    Cell Death and Disease, Vol. 1, Núm. 7

2009

  1. Endoplasmic reticulum Ca2+ release through ryanodine and IP3 receptors contributes to neuronal excitotoxicity

    Cell Calcium, Vol. 46, Núm. 4, pp. 273-281