Publicacions en col·laboració amb investigadors/es de Instituto Biofisika (14)

2024

  1. Fluorometric Measurement of Calmodulin-Dependent Peptide–Protein Interactions Using Dansylated Calmodulin

    Bio-protocol, Vol. 14, Núm. 7

  2. Identification of Riluzole derivatives as novel calmodulin inhibitors with neuroprotective activity by a joint synthesis, biosensor, and computational guided strategy

    Biomedicine and Pharmacotherapy, Vol. 174

  3. MLe-KCNQ2: An Artificial Intelligence Model for the Prognosis of Missense KCNQ2 Gene Variants

    International Journal of Molecular Sciences, Vol. 25, Núm. 5

2023

  1. Molecular dynamics simulations of the calmodulin-induced α-helix in the SK2 calcium-gated potassium ion channel

    Journal of Biological Chemistry, Vol. 299, Núm. 2

  2. Redox regulation of KV7 channels through EF3 hand of calmodulin

    eLife, Vol. 12

2021

  1. An epilepsy-causing mutation leads to co-translational misfolding of the Kv7.2 channel

    BMC Biology, Vol. 19, Núm. 1

  2. Do calmodulin binding IQ motifs have built-in capping domains?

    Protein Science, Vol. 30, Núm. 10, pp. 2029-2041

2018

  1. Calmodulin: A multitasking protein in Kv7.2 potassium channel functions

    Biomolecules, Vol. 8, Núm. 3

  2. Lack of correlation between surface expression and currents in epileptogenic ab-calmodulin binding domain kv7.2 potassium channel mutants

    Channels, Vol. 12, Núm. 1, pp. 299-310

2017

  1. Calmodulin confers calcium sensitivity to the stability of the distal intracellular assembly domain of Kv7.2 channels

    Scientific Reports, Vol. 7, Núm. 1

  2. Differential regulation of pi(4,5)p2 sensitivity of kv7.2 and kv7.3 channels by calmodulin

    Frontiers in Molecular Neuroscience, Vol. 10

2015

  1. Uncoupling PIP2-calmodulin regulation of Kv7.2 channels by an assembly destabilizing epileptogenic mutation

    Journal of Cell Science, Vol. 128, Núm. 21, pp. 4014-4023

2011

  1. Kv7 channels can function without constitutive calmodulin tethering

    PLoS ONE, Vol. 6, Núm. 9

2008

  1. A novel KCNQ4 pore-region mutation (p.G296S) causes deafness by impairing cell-surface channel expression

    Human Genetics, Vol. 123, Núm. 1, pp. 41-53